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β tc 6 mouse pancreatic β  (ATCC)


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    ATCC β tc 6 mouse pancreatic β
    β Tc 6 Mouse Pancreatic β, supplied by ATCC, used in various techniques. Bioz Stars score: 95/100, based on 80 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/6+mouse+pancreatic+%CE%B2+cells/pm40594521-254-25-32?v=ATCC
    Average 95 stars, based on 80 article reviews
    β tc 6 mouse pancreatic β - by Bioz Stars, 2026-07
    95/100 stars

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    A ROS may result in ER-stress. ROS or/and ER-stress incurs JNK activation. The activated JNK leads to <t>pancreatic</t> <t>β</t> <t>cell</t> apoptosis via mitochondria-dependent or independent pathways. B Acute ROS or ER-stress results in the expression of NR4A1, which in turn enhances the expression of MKP7, while MKP7 is able to abolish partial JNK activation by exerting its phosphatase activity, therefore, largely reduces pancreatic β cell apoptosis. The fate of pancreatic β cells depends on the balance between the availability of NR4A1 or MKP7 and the severity of ROS or ER-stress.
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    A ROS may result in ER-stress. ROS or/and ER-stress incurs JNK activation. The activated JNK leads to pancreatic β cell apoptosis via mitochondria-dependent or independent pathways. B Acute ROS or ER-stress results in the expression of NR4A1, which in turn enhances the expression of MKP7, while MKP7 is able to abolish partial JNK activation by exerting its phosphatase activity, therefore, largely reduces pancreatic β cell apoptosis. The fate of pancreatic β cells depends on the balance between the availability of NR4A1 or MKP7 and the severity of ROS or ER-stress.

    Journal: Cell Death Discovery

    Article Title: NR4A1 enhances MKP7 expression to diminish JNK activation induced by ROS or ER-stress in pancreatic β cells for surviving

    doi: 10.1038/s41420-021-00521-0

    Figure Lengend Snippet: A ROS may result in ER-stress. ROS or/and ER-stress incurs JNK activation. The activated JNK leads to pancreatic β cell apoptosis via mitochondria-dependent or independent pathways. B Acute ROS or ER-stress results in the expression of NR4A1, which in turn enhances the expression of MKP7, while MKP7 is able to abolish partial JNK activation by exerting its phosphatase activity, therefore, largely reduces pancreatic β cell apoptosis. The fate of pancreatic β cells depends on the balance between the availability of NR4A1 or MKP7 and the severity of ROS or ER-stress.

    Article Snippet: The mouse pancreatic β cell lines (MIN6 and β-TC6) were from ATCC.

    Techniques: Activation Assay, Expressing, Activity Assay